Free NAPLEX – North American Pharmacist Licensure Examination Practice Test Question

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  50ml/hr

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  5ml/hr

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  5ml/min

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  0.5ml/min

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  7ml/hr
  
  

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  Amitriptyline

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  Nortriptyline

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  Doxepin

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  Fluvoxamine

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  Escitalopram
  
  

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  Glucuronidation

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  Sulfation

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  Cytochrome P-450 oxidation

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  Direct renal excretion
  The major route is glucuronidation catalyzed by UDP-glucuronyl transferase in the liver. Sulfation is the next most common route and is the target mechanism for NAC therapy. Oxidation by cytochrome P-450 results in the formation of N-acetyl-p-benzoquinone imine, which is responsible for the hepatic necrosis caused by acetaminophen overdose. Direct renal excretion represents approximately 5% of the metabolism of acetaminophen. Plasma metabolism of acetaminophen does not generally occur.
  
  

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  Plasma breakdown
  
  

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  Dapsone is bacteriostatic because of its inhibitory effects on dihydrofolate reductase

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  Dapsone is bacteriostatic because of its inhibitory effects on myeloperoxidase

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  Clofazimine is bactericidal by directly inhibiting bacterial DNA polymerase
  

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  Rifampin is bacteriostatic by inhibiting RNA synthesis by blocking DNA-dependent RNA polymerase

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  Rifampin is bactericidal by inhibiting RNA synthesis by blocking DNA-dependent RNA polymerase
  
  

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  Acarbose

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  Metformin

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  Dapagliflozin

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  Pioglitazone

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  Sitagliptin
  
  

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  1 gram

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  8 grams

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  12 grams

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  14 grams
  
  

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  16 units of insulin glargine once daily, Insulin Lispro 4 units 3 times a day with meals

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  15 units of insulin glargine once daily, Insulin Lispro 5 units 3 times a day with meals

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  23 units of insulin glargine once daily, Insulin Lispro 7 units 3 times a day with meals

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  30 units of insulin glargine once daily, Insulin Lispro 6 units 3 times a day with meals
  

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  18 units of insulin glargine once daily, Insulin Lispro 6 units 3 times a day with meals
  
  

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  0.9 kcal/g
  

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  1.2 kcal/g
  

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  1.5 kcal/g
  

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  3.4 kcal/g
  
  
  

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  Sumatriptan
  Migraine headaches typically affect females more often than males, and patients most frequently present in their early 20s. Classic symptoms of migraine include throbbing headaches lasting between 2–24 hours in duration, with triggers such as red wine, fasting, stress, and menses. Primary prevention is aimed at the identification and avoidance of triggers. Over the counter NSAIDS can be used if symptoms persist. Failing this, PRN abortive therapy is indicated, including the triptans (e.g. – sumatriptan) and metoclopramide. Choice B – Gabapentin is an anticonvulsant that is considered to be a second-line, prophylactic treatment for recurrent migraine headaches. Its utility is limited by its lengthy side effect profile. Choice C – Amitriptyline, a tricyclic antidepressant, can also be utilized for migraine prophylaxis. However, it will not abort a migraine currently in progress, and extensive side effects limit its use. Choices D + E – Propranolol and diltiazam are beta-blockers and calcium channel blockers, respectively. As with the anticonvulsants and tricyclic antidepressants, these are considered migraine prophylaxis and will not interrupt a migraine once it has begun.
  
  

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  Gabapentin

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  Amitriptyline

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  Propranolol

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  Diltiazam
  
  

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  Sulfonylureas

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  Alpha- Glucosidase Inhibitors

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  DPP4 Inhibitors

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  Glucagon-like peptide-1 receptor agonists

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  Thiazolidinediones

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  Biguanide

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  SGLT2 inhibitors